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Mandatory Fields
James, JE,Gregg, MED,Matyas, TA,Hughes, BM,Howard, S
2012
May
Biological Psychology
Stress reactivity and the Hemodynamic Profile-Compensation Deficit (HP-CD) Model of blood pressure regulation
Published
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Optional Fields
Blood pressure Reactivity Stress Cardiovascular disease Cardiac output Peripheral resistance Hemodynamic profile Compensation deficit CORONARY-HEART-DISEASE ENHANCED CAROTID ATHEROSCLEROSIS PERIPHERAL VASCULAR-RESPONSES ACUTE EMOTIONAL-STRESS TRADE-CENTER ATTACK AGED FINNISH MEN CARDIOVASCULAR REACTIVITY INDIVIDUAL-DIFFERENCES PSYCHOLOGICAL STRESS RISK-FACTOR
90
161
170
Considering the central role of the concept of blood pressure reactivity to explanations of the influence of life stress in the pathogenesis of cardiovascular disease, it is important that the underlying psychophysiological determinants of blood pressure change are elucidated. Empirical evidence is reviewed concerning the utility of the Hemodynamic Profile-Compensation Deficit (HP-CD) Model (Gregg et al., 2002), which draws on physiological theory that explains blood pressure regulation as a dynamic compensatory relation between cardiac output and total peripheral resistance. Core constructs and quantitative features of the Model are explained, accompanied by a focussed review of the Model's application in reactivity studies of stress, lifestyle behaviours, laboratory-to-field generalisability, and personality differences. Although the available studies are relatively few in number, evidence of the Model's utility is promising. Application of the Model in further research could help to elucidate cardiovascular pathogenesis in ways not achievable through the study of blood pressure reactivity alone. (C) 2012 Elsevier B.V. All rights reserved.
DOI 10.1016/j.biopsycho.2012.02.021
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